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So as you would expect and at anytime you do a stress study like this you would expect a linear increase.
大家可以想象不管什么时候 当你做一个这样的压力研究时你都可以看到
In these pro-inflammatory cytokines as what you get traditionally And that's definitely what we've found.
这些前炎性细胞因子呈线性增加的趋势这和传统结论是一致的我们的结论也是如此
So this isn't looking at any other factors this is just on average individuals showed an increase in IL-6 in response to the acute stressor.
我们没有考虑其他因素一般人只是显示出在面对短期压力时IL-6水平的上升
And the very same thing is seen for the IL-6 to IL-10 This is significant time effect Then we found that poor sleep.
而且IL-6和IL-10的比例也出现了相同的变化这是一个重要的时间效应然后我们发现睡眠不好的情况
But not BMI these were looked at separately potentiated IL-6.
没有考虑身体质量指数这些因素都是分别考察的在面对短期压力时 睡眠不好会影响到
And IL-6/IL-10 responses to acute stress So here you see this So you get this kind of graded effect.
IL-6水平和IL-6和IL-10的比例大家看看这张图这里有一个梯度效应
That good sleepers had the least slope Average sleepers had a steeper increase but the poor sleepers had a very strong high increase.
睡眠良好的人坡度最缓睡眠一般的人坡度稍陡而睡眠不好的人坡度上升最快
Suggesting that poor sleep is in some way operating to moderate the effect of this stressor on biology specifically in this case it's IL-6/IL-10. But it's the same for IL-6.
这说明了 睡眠不好以某种方式调节了压力对生物机能的影响特别是IL-6和IL-10的比例, 但对IL-6的影响也是一样的.
But interestingly it was the combination between BMI and sleep.
然而有趣的是只有当BMI和睡眠结合起来时
That was the worst So here we see poor sleepers with low BMI had the weak slope.
才会出现最糟情况这里大家可以看到BMI指数低睡眠质量不好的人坡度较缓
The good sleepers and this is... Is broken down by BMI because the interaction between good sleepers and BMI was non-significant.
而睡眠质量好的人… BMI在这里发生了失效因为如果睡眠质量好和BMI一般那么它们之间的关系并不显着
